Introduction
Vitamin D deficiency is a prevalent, modifiable determinant of poor musculoskeletal health. Public health interventions have aimed to improve population vitamin D status. However, there is minimal longitudinal data assessing whether these programs have improved vitamin D sufficiency or evaluating the skeletal health outcomes of maintaining or achieving vitamin D sufficiency.
Methods
Community-dwelling adults aged 50-80 years had 25-hydroxyvitamin D (25(OH)D) assessed by radioimmunoassay at baseline (n=1096), 2.5 (n=870) and 10 (n=565) years in a Tasmanian population-based prospective cohort study. Sun exposure was quantified by questionnaire, supplement use at clinic review and BMD by DXA. 25(OH)D <50nmol/L was considered deficient.
Results
Over 10 years mean 25(OH)D increased (52.4±18.7 to 62.5±23.5 nmol/L, p<0.001) and the percentage of vitamin D deficient participants decreased (48.5% to 29.8%, p<0.001). Participants with baseline deficiency had larger 25(OH)D increases than baseline sufficient participants (19.2±25.3 vs 1.6±23.3 nmol/L, p<0.001). The proportion of participants taking vitamin D supplements increased (2.3% to 37.1%, p<0.001). Longitudinal change in 25(OH)D was associated with baseline summer (β=1.46, p<0.001) and winter (β=1.29, p=0.003) sun exposure, change in summer (β=1.27, p=0.002) and winter (β=1.47, p<0.001) sun exposure and vitamin D supplement use (β=24.9-34.8, p<0.001). Participants who were always vitamin D sufficient retained significantly more BMD at all sites (β=0.019 – 0.030, p=0.006 – 0.047 vs always deficient group). Participants who achieved vitamin D sufficiency had greater BMD gains at the lumbar spine (β=0.019, p=0.02) and similar BMD outcomes elsewhere compared to those who were always sufficient.
Conclusions
Cohort 25(OH)D concentration increased and vitamin D sufficiency was associated with less BMD loss over 10 years. This suggests that (1) intervention can improve population vitamin D status, (2) vitamin D sufficiency may ameliorate BMD loss in older adults and (3) vitamin D repletion of deficient individuals can achieve similar longitudinal BMD outcomes as persistent sufficiency.